In the last few decades we have understood a major concept in the genesis of cardiac arrhythmia.Slowing in the propagation of cardiac impulse is a key trigger to precipitate a reentry circuit and initiate a tachy- arrhythmia.Still , many conditions like first degree AV block, chronic RBBB or even LBBB are benign entities as along as the heart is structurally normal .They seem never increase the incidence or life time risk of cardiac arrhythmia . Longevity is unaffected.( Or do we assume many things ?)
How is this possible ? or is the theory of slow conduction triggering reentry is flawed ?
Think again . . . if these patients who later on develop a structural heart disease , with an episode of ACS , myocardial or valvular disease, the original slow conduction substrates these people were harboring , will it become important ?
Surprisingly , we have no answers in literature.When Haissaguerre et al found preexisting ERS pattern could be a trigger for primary VF in case they develop ACS , he opened up a huge debate as it involved converting a vast number of normal population electrically anxious.
Now ,is it possible the so called benign blocks of heart like first degree AV blocks , RBBB , LAHBa , would be important at times of ACS and possibly make them prone for for primary ischemic arrhythmia .
Is bundle branch re-entry possible in structurally normal heart ?
We need answers. Some one , (Any EP fellow) somewhere could take up the issue and enlighten us !